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neutralizing antibodies against cxcl10  (R&D Systems)


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    R&D Systems neutralizing antibodies against cxcl10
    CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and <t>CXCL10.</t> Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05
    Neutralizing Antibodies Against Cxcl10, supplied by R&D Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/neutralizing antibodies against cxcl10/product/R&D Systems
    Average 90 stars, based on 1 article reviews
    neutralizing antibodies against cxcl10 - by Bioz Stars, 2026-05
    90/100 stars

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    1) Product Images from "Tuberculous pleural effusion-induced Arg-1 + macrophage polarization contributes to lung cancer progression via autophagy signaling"

    Article Title: Tuberculous pleural effusion-induced Arg-1 + macrophage polarization contributes to lung cancer progression via autophagy signaling

    Journal: Respiratory Research

    doi: 10.1186/s12931-024-02829-8

    CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and CXCL10. Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05
    Figure Legend Snippet: CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and CXCL10. Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05

    Techniques Used: Recombinant, Cell Culture, Binding Assay

    Schema of mechanism by which TPE-induced Arg-1 + macrophage polarization contributes to lung cancer malignancy. CXCL9 and CXCL10 are involved in TPE-induced macrophage M2 Arg-1 + polarization. TPE-Arg-1 + MФ CM induces A549 proliferation by upregulating autophagy and E-cadherin signaling. ABH reversed the effect of TPE-Arg-1 + MФ CM leading to cancer growth suppression by downregulating autophagy and E-cadherin signaling. ABH: 2(S)-amino-6-boronohexanoic acid (an arginase inhibitor)
    Figure Legend Snippet: Schema of mechanism by which TPE-induced Arg-1 + macrophage polarization contributes to lung cancer malignancy. CXCL9 and CXCL10 are involved in TPE-induced macrophage M2 Arg-1 + polarization. TPE-Arg-1 + MФ CM induces A549 proliferation by upregulating autophagy and E-cadherin signaling. ABH reversed the effect of TPE-Arg-1 + MФ CM leading to cancer growth suppression by downregulating autophagy and E-cadherin signaling. ABH: 2(S)-amino-6-boronohexanoic acid (an arginase inhibitor)

    Techniques Used:



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    neutralizing antibodies against cxcl10  (R&D Systems)
    90
    R&D Systems neutralizing antibodies against cxcl10
    CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and <t>CXCL10.</t> Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05
    Neutralizing Antibodies Against Cxcl10, supplied by R&D Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/neutralizing antibodies against cxcl10/product/R&D Systems
    Average 90 stars, based on 1 article reviews
    neutralizing antibodies against cxcl10 - by Bioz Stars, 2026-05
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    neutralizing antibody against cxcl10 gtx31179  (GeneTex)
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    CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and <t>CXCL10.</t> Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05
    Neutralizing Antibody Against Cxcl10 Gtx31179, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/neutralizing antibody against cxcl10 gtx31179/product/GeneTex
    Average 90 stars, based on 1 article reviews
    neutralizing antibody against cxcl10 gtx31179 - by Bioz Stars, 2026-05
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    Image Search Results


    CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and CXCL10. Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05

    Journal: Respiratory Research

    Article Title: Tuberculous pleural effusion-induced Arg-1 + macrophage polarization contributes to lung cancer progression via autophagy signaling

    doi: 10.1186/s12931-024-02829-8

    Figure Lengend Snippet: CXL9 and CXL10 in TPE modulate M2 macrophage polarization. a Cytokine array analysis demonstrated differences between TPE and T. Spots with differentially regulated cytokines are identified with a red box and a blue box. Red boxes correspond to CD30, CXCL9, and CXCL10. Blue boxes correspond to ApoA1, YKL-40, IGFBP-2, and IGFBP-3. b Mouse BMDMs were treated with 20 µg recombinant CXCL9 and 20 µg recombinant CXCL10. c Mouse BMDMs were cultured with neutralizing antibodies for CXCL9 (20 µg) and CXCL10 (20 µg) after TPE treatment. Arg-1 mRNA, Ym-1 mRNA, and CXCR3 mRNA levels were measured. ApoA1: apolipoprotein A1; YKL-40: chitinase-3-like protein 1; IGFBP-2: insulin like growth factor binding protein 2; IGFBP-3: insulin like growth factor binding protein 3. The values represent the results of three experiments. ** p < 0.01, * p < 0.05

    Article Snippet: Recombinant CXCL9 and CXCL10 and neutralizing antibodies against CXCL9 and CXCL10 were purchased from R&D Systems.

    Techniques: Recombinant, Cell Culture, Binding Assay

    Schema of mechanism by which TPE-induced Arg-1 + macrophage polarization contributes to lung cancer malignancy. CXCL9 and CXCL10 are involved in TPE-induced macrophage M2 Arg-1 + polarization. TPE-Arg-1 + MФ CM induces A549 proliferation by upregulating autophagy and E-cadherin signaling. ABH reversed the effect of TPE-Arg-1 + MФ CM leading to cancer growth suppression by downregulating autophagy and E-cadherin signaling. ABH: 2(S)-amino-6-boronohexanoic acid (an arginase inhibitor)

    Journal: Respiratory Research

    Article Title: Tuberculous pleural effusion-induced Arg-1 + macrophage polarization contributes to lung cancer progression via autophagy signaling

    doi: 10.1186/s12931-024-02829-8

    Figure Lengend Snippet: Schema of mechanism by which TPE-induced Arg-1 + macrophage polarization contributes to lung cancer malignancy. CXCL9 and CXCL10 are involved in TPE-induced macrophage M2 Arg-1 + polarization. TPE-Arg-1 + MФ CM induces A549 proliferation by upregulating autophagy and E-cadherin signaling. ABH reversed the effect of TPE-Arg-1 + MФ CM leading to cancer growth suppression by downregulating autophagy and E-cadherin signaling. ABH: 2(S)-amino-6-boronohexanoic acid (an arginase inhibitor)

    Article Snippet: Recombinant CXCL9 and CXCL10 and neutralizing antibodies against CXCL9 and CXCL10 were purchased from R&D Systems.

    Techniques: